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GPR35 agonists (YE120, zaprinast, and pamoic acid) promoted wound fix in the focus-dependent fashion independently of mobile proliferation, whereas a specific GPR35 antagonist CID2745687, forskolin, and pertussis toxin reversed the YE120-induced influence. YE120 improved the mRNA expression of fibronectin and its receptor integrin α5, and ERK1/2 p

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